Congenital Asplenia in Mice and Humans with Mutations in a Pbx/Nkx2-5/p15 Module
Open Access
- 15 May 2012
- journal article
- research article
- Published by Elsevier BV in Developmental Cell
- Vol. 22 (5), 913-926
- https://doi.org/10.1016/j.devcel.2012.02.009
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- A Conserved Pbx-Wnt-p63-Irf6 Regulatory Module Controls Face Morphogenesis by Promoting Epithelial ApoptosisDevelopmental Cell, 2011
- Cooperative Transcriptional Activation by Klf4, Meis2, and Pbx1Molecular and Cellular Biology, 2011
- Gain-of-function human STAT1 mutations impair IL-17 immunity and underlie chronic mucocutaneous candidiasisThe Journal of Experimental Medicine, 2011
- Whole-Exome-Sequencing-Based Discovery of Human FADD DeficiencyAmerican Journal of Human Genetics, 2010
- Whole-exome sequencing-based discovery of STIM1 deficiency in a child with fatal classic Kaposi sarcomaThe Journal of Experimental Medicine, 2010
- The Genome Analysis Toolkit: A MapReduce framework for analyzing next-generation DNA sequencing dataGenome Research, 2010
- Fast and accurate short read alignment with Burrows–Wheeler transformBioinformatics, 2009
- An Nkx2-5/Bmp2/Smad1 Negative Feedback Loop Controls Heart Progenitor Specification and ProliferationCell, 2007
- Limited overlapping roles of P15INK4b and P18INK4c cell cycle inhibitors in proliferation and tumorigenesisThe EMBO Journal, 2000
- Cre–mediated chromosome loss in miceNature Genetics, 1997