Tumor Necrosis Factor-α Selectively InducesMnSOD Expression via Mitochondria-to-Nucleus Signaling, whereas Interleukin-1β Utilizes an Alternative Pathway
Open Access
- 1 January 2001
- journal article
- Published by Elsevier BV
- Vol. 276 (23), 20419-20427
- https://doi.org/10.1074/jbc.m008915200
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- Cells depleted of mitochondrial DNA (p0) yield insight into physiological mechanismsFEBS Letters, 1999
- Novel Inhibitors of Cytokine-induced IκBα Phosphorylation and Endothelial Cell Adhesion Molecule Expression Show Anti-inflammatory Effects in VivoJournal of Biological Chemistry, 1997
- Conditions allowing redox-cycling ubisemiquinone in mitochondria to establish a direct redox couple with molecular oxygenFree Radical Biology & Medicine, 1996
- p38 Mitogen-activated Protein Kinase Phosphorylates Cytosolic Phospholipase A2 (cPLA2) in Thrombin-stimulated PlateletsPublished by Elsevier BV ,1996
- The Oxidative Metabolism of GlutamineJournal of Biological Chemistry, 1996
- Stimulation of synaptosomal free radical production by fatty acids: Relation to esterification and to degree of unsaturationFEBS Letters, 1995
- Down-regulation of Tumor Necrosis Factor Receptors by Blockade of Mitochondrial RespirationPublished by Elsevier BV ,1995
- SUPEROXIDE RADICAL AND SUPEROXIDE DISMUTASESAnnual Review of Biochemistry, 1995
- Function and Activation of NF-kappaB in the Immune SystemAnnual Review of Immunology, 1994
- Regulation of manganese superoxide dismutase: IL-1 and TNF induction in pulmonary artery and microvascular endothelial cellsBiochemical and Biophysical Research Communications, 1992