Multiple proteins implicated in neurodegenerative diseases accumulate in axons after brain trauma in humans
- 10 July 2007
- journal article
- Published by Elsevier BV in Experimental Neurology
- Vol. 208 (2), 185-192
- https://doi.org/10.1016/j.expneurol.2007.06.018
Abstract
No abstract availableKeywords
This publication has 70 references indexed in Scilit:
- Caspase inhibition therapy abolishes brain trauma-induced increases in Aβ peptide: Implications for clinical outcomeExperimental Neurology, 2006
- Alzheimer's pathology in human temporal cortex surgically excised after severe brain injuryExperimental Neurology, 2004
- Axonal Damage in Traumatic Brain InjuryThe Neuroscientist, 2000
- Regulation of APP cleavage by α‐, β‐ and γ‐secretasesFEBS Letters, 2000
- Quantification of Axonal Damage in Traumatic Brain InjuryJournal of Neurochemistry, 1999
- Sudden Unexpected Death in Epilepsy: Terminology and DefinitionsEpilepsia, 1997
- Alzheimer's disease after remote head injury: an incidence study.Journal of Neurology, Neurosurgery & Psychiatry, 1997
- Rapid appearance of β-amyloid precursor protein immunoreactivity in damaged axons and reactive glial cells in rat brain following needle stab injuryBrain Research, 1991
- Neuropathological stageing of Alzheimer-related changesActa Neuropathologica, 1991
- Posttraumatic ParkinsonismSurgical Neurology, 1985