Mutations in the Transmembrane Domain of APP Altering γ-Secretase Specificity
- 1 December 1997
- journal article
- research article
- Published by American Chemical Society (ACS) in Biochemistry
- Vol. 36 (49), 15396-15403
- https://doi.org/10.1021/bi971071m
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- The “Nonamyloidogenic” p3 Fragment (Amyloid β17-42) Is a Major Constituent of Down's Syndrome Cerebellar PreamyloidPublished by Elsevier BV ,1996
- β-Amyloid Precursor ProteinOnline Journal of Public Health Informatics, 1996
- Alzheimer's Disease βA4 Protein Release and Amyloid Precursor Protein Sorting Are Regulated by Alternative SplicingPublished by Elsevier BV ,1996
- Amyloidogenic processing of the human amyloid precursor protein in primary cultures of rat hippocampal neuronsJournal of Neuroscience, 1996
- Amyloid β Protein (Aβ) in Alzheimeri's Disease BrainPublished by Elsevier BV ,1995
- Cell Biology of the Amyloid beta-Protein Precursor and the Mechanism of Alzheimer's DiseaseAnnual Review of Cell Biology, 1994
- Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43)Neuron, 1994
- Generation of βA4 from the amyloid protein precursor and fragments thereofFEBS Letters, 1993
- Membrane inserted APP fragments containing the βA4 sequence of Alzheimer's disease do not aggregateFEBS Letters, 1992
- PYRUVOYL-DEPENDENT ENZYMESAnnual Review of Biochemistry, 1990