Human ARF binds E2F1 and inhibits its transcriptional activity
- 1 March 2001
- journal article
- Published by Springer Science and Business Media LLC in Oncogene
- Vol. 20 (9), 1033-1041
- https://doi.org/10.1038/sj.onc.1204220
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 35 references indexed in Scilit:
- Two arginine rich domains in the p14ARF tumour suppressor mediate nucleolar localizationOncogene, 2000
- An N-terminal p14ARF peptide blocks Mdm2-dependent ubiquitination in vitro and can activate p53 in vivoOncogene, 2000
- Inactivation of the INK4A/ARF locus frequently coexists with TP53 mutations in non-small cell lung cancerOncogene, 1999
- Signaling to p53Cell, 1998
- The INK4a/ARF tumor suppressor: one gene—two products—two pathwaysTrends in Biochemical Sciences, 1998
- The human protein p19ARF is not detected in hemopoietic human cell lines that abundantly express the alternative β transcript of the p16INK4a/MTS1 geneOncogene, 1997
- E2F1-induced apoptosis requires DNA binding but not transactivation and is inhibited by the retinoblastoma protein through direct interaction.Genes & Development, 1997
- Stimulation of E2F1/DP1 transcriptional activity by MDM2 oncoproteinNature, 1995
- A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4Nature, 1993
- E2F: a Link Between the Rb Tumor Suppressor Protein and Viral OncoproteinsScience, 1992