RANKL-stimulated osteoclast-like cell formation in vitro is partially dependent on endogenous interleukin-1 production
- 31 May 2006
- journal article
- Published by Elsevier BV in Bone
- Vol. 38 (5), 678-685
- https://doi.org/10.1016/j.bone.2005.10.011
Abstract
No abstract availableThis publication has 51 references indexed in Scilit:
- Bidirectional signaling between stromal and hemopoietic cells regulates interleukin-1 expression during human osteoclast formationBone, 1999
- Interleukin 1 Induces Multinucleation and Bone-Resorbing Activity of Osteoclasts in the Absence of Osteoblasts/Stromal CellsExperimental Cell Research, 1999
- Mice Lacking the Type I Interleukin-1 Receptor Do Not Lose Bone Mass after OvariectomyEndocrinology, 1998
- Activation of NF-κB Is Involved in the Survival of Osteoclasts Promoted by Interleukin-1Online Journal of Public Health Informatics, 1998
- Estrogen Deficiency Increases the Ability of Stromal Cells to Support Murine Osteoclastogenesis via an Interleukin-1and Tumor Necrosis Factor-mediated Stimulation of Macrophage Colony-stimulating Factor ProductionOnline Journal of Public Health Informatics, 1996
- Osteoclast function is activated by osteoblastic cells through a mechanism involving cell-to-cell contact.Endocrinology, 1996
- Simultaneous block of interleukin-1 and tumor necrosis factor is required to completely prevent bone loss in the early postovariectomy periodEndocrinology, 1995
- Macrophage colony-stimulating factor and interleukin-1 alpha maintain the survival of osteoclast-like cellsEndocrinology, 1995
- Interleukin-1 receptor antagonist and tumor necrosis factor binding protein decrease osteoclast formation and bone resorption in ovariectomized mice.JCI Insight, 1994
- Stimulation of prostaglandin E2 and bone resorption by recombinant human interleukin 1 alpha in fetal mouse bonesBiochemical and Biophysical Research Communications, 1986