Advanced Glycation End Products Induce Tubular Epithelial-Myofibroblast Transition through the RAGE-ERK1/2 MAP Kinase Signaling Pathway
- 1 April 2004
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 164 (4), 1389-1397
- https://doi.org/10.1016/s0002-9440(10)63225-7
Abstract
No abstract availableThis publication has 48 references indexed in Scilit:
- Functional genomic analysis of the ADP‐ribosylation factor family of GTPases: phylogeny among diverse eukaryotes and function inC. elegansThe FASEB Journal, 2004
- Advanced glycation end products activate Smad signaling via TGF‐β‐dependent and ‐independent mechanisms: implications for diabetic renal and vascular diseaseThe FASEB Journal, 2003
- Receptor for Advanced Glycation End Products (RAGE) Signaling Induces CREB-dependent Chromogranin Expression during Neuronal DifferentiationPublished by Elsevier BV ,2002
- Smad7 Inhibits Fibrotic Effect of TGF-β on Renal Tubular Epithelial Cells by Blocking Smad2 ActivationJournal of the American Society of Nephrology, 2002
- Diabetic nephropathy: It's in the numbersKidney International, 2002
- Mitogen-Activated Protein (MAP) Kinase Pathways: Regulation and Physiological FunctionsEndocrine Reviews, 2001
- The tubulointerstitium in progressive diabetic kidney disease: More than an aftermath of glomerular injury?Kidney International, 1999
- Transforming growth factor-β regulates tubular epithelial-myofibroblast transdifferentiation in vitroKidney International, 1999
- PDGF and TGF-β mediate collagen production by mesangial cells exposed to advanced glycosylation end productsKidney International, 1995
- A novel, simple, reliable, and sensitive method for multiple immunoenzyme staining: use of microwave oven heating to block antibody crossreactivity and retrieve antigens.Journal of Histochemistry & Cytochemistry, 1995