Reduced efficiency and increased mutagenicity of translesion DNA synthesis across a TT cyclobutane pyrimidine dimer, but not a TT 6-4 photoproduct, in human cells lacking DNA polymerase η
- 1 October 2008
- journal article
- Published by Elsevier BV in DNA Repair
- Vol. 7 (10), 1636-1646
- https://doi.org/10.1016/j.dnarep.2008.06.008
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Lesion bypass in yeast cells: Pol eta participates in a multi-DNA polymerase processThe EMBO Journal, 2002
- Quantitative measurement of translesion replication in human cells: Evidence for bypass of abasic sites by a replicative DNA polymeraseProceedings of the National Academy of Sciences of the United States of America, 2002
- DNA Double-Strand Break Repair by Homologous RecombinationBiological Chemistry, 2002
- Cyclobutane Pyrimidine Dimers Are Responsible for the Vast Majority of Mutations Induced by UVB Irradiation in Mammalian CellsJournal of Biological Chemistry, 2001
- The Y-Family of DNA PolymerasesMolecular Cell, 2001
- Genome maintenance mechanisms for preventing cancerNature, 2001
- DNA double-strand breaks: signaling, repair and the cancer connectionNature Genetics, 2001
- Response of human DNA polymerase iota to DNA lesionsNucleic Acids Research, 2001
- Polymerase η deficiency in the xeroderma pigmentosum variant uncovers an overlap between the S phase checkpoint and double-strand break repairProceedings of the National Academy of Sciences of the United States of America, 2000
- Fidelity of Human DNA Polymerase ηJournal of Biological Chemistry, 2000