Loxosceles Sphingomyelinase Induces Complement-Dependent Dermonecrosis, Neutrophil Infiltration, and Endogenous Gelatinase Expression
- 1 April 2005
- journal article
- Published by Elsevier BV in Journal of Investigative Dermatology
- Vol. 124 (4), 725-731
- https://doi.org/10.1111/j.0022-202x.2005.23654.x
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Molecular cloning, expression, function and immunoreactivities of members of a gene family of sphingomyelinases from Loxosceles venom glandsMolecular Immunology, 2004
- Loxosceles spider venom induces metalloproteinase mediated cleavage of MCP/CD46 and MHCI and induces protection against C‐mediated lysisImmunology, 2002
- Sphingomyelinases in the Venom of the SpiderLoxosceles intermediaAre Responsible for both Dermonecrosis and Complement-Dependent HemolysisBiochemical and Biophysical Research Communications, 1998
- Attenuation of Interleukin-8 Expression in C6-deficient Rabbits After Myocardial Ischemia/ReperfusionJournal of Molecular and Cellular Cardiology, 1998
- Neutrophil Elastase Processing of Gelatinase A Is Mediated by Extracellular MatrixBiochemistry, 1995
- Wound Fluid from Chronic Leg Ulcers Contains Elevated Levels of Metalloproteinases MMP-2 and MMP-9Journal of Investigative Dermatology, 1993
- LoxoscelismThe American Journal of the Medical Sciences, 1992
- Loxoscelismo en Chile: estudios epidemiológicos, clínicos y experimentalesRevista do Instituto de Medicina Tropical de São Paulo, 1989
- Loxoscelism and Necrotic ArachnidismJournal of Toxicology: Clinical Toxicology, 1983
- Biology and Venoms of the Most Important Soutli American Spiders of the GeneraPhoneutria, Loxosceles, Lycosa, andLatrodectusAmerican Zoologist, 1969