Peroxisome Proliferator-activated Receptor α Is Responsible for the Up-regulation of Hepatic Glucose-6-phosphatase Gene Expression in Fasting and db/db Mice
Open Access
- 1 January 2011
- journal article
- Published by Elsevier BV
- Vol. 286 (2), 1157-1164
- https://doi.org/10.1074/jbc.m110.157875
Abstract
No abstract availableThis publication has 61 references indexed in Scilit:
- Interrelationship between Liver X Receptor α, Sterol Regulatory Element-binding Protein-1c, Peroxisome Proliferator-activated Receptor γ, and Small Heterodimer Partner in the Transcriptional Regulation of Glucokinase Gene Expression in LiverJournal of Biological Chemistry, 2009
- International Union of Pharmacology. LXI. Peroxisome Proliferator-Activated ReceptorsPharmacological Reviews, 2006
- Pancreatic islet response to hyperglycemia is dependent on peroxisome proliferator‐activated receptor alpha (PPARα)FEBS Letters, 2005
- Regulation of Glucose-6-phosphatase Gene Expression in Cultured Hepatocytes and H4IIE Cells by Short-chain Fatty AcidsPublished by Elsevier BV ,2003
- Dexamethasone induction of hypertension and diabetes is PPAR-α dependent in LDL receptor–null miceNature Medicine, 2003
- Hepatocyte nuclear factor-4alpha mediates the stimulatory effect of peroxisome proliferator-activated receptor gamma co-activator-1alpha (PGC-1alpha) on glucose-6-phosphatase catalytic subunit gene transcription in H4IIE cellsBiochemical Journal, 2003
- Peroxisome Proliferator-activated Receptor α (PPARα) Turnover by the Ubiquitin-Proteasome System Controls the Ligand-induced Expression Level of Its Target GenesPublished by Elsevier BV ,2002
- Peroxisome proliferator–activated receptor α mediates the adaptive response to fastingJCI Insight, 1999
- The effects of free fatty acids on gluconeogenesis and glycogenolysis in normal subjectsJCI Insight, 1999
- Isolation of a cDNA for the Catalytic Subunit of Rat Liver Glucose-6-Phosphatase: Regulation of Gene Expression in FAO Hepatoma Cells by Insulin, Dexamethasone and cAMPBiochemical and Biophysical Research Communications, 1994