The immunosuppressant FK506 increases GAP-43 mRNA levels in axotomized sensory neurons
- 1 January 1998
- journal article
- Published by Elsevier BV in Neuroscience Letters
- Vol. 241 (1), 25-28
- https://doi.org/10.1016/s0304-3940(97)00960-9
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- FK506 and the role of immunophilins in nerve regenerationMolecular Neurobiology, 1997
- Targeted Overexpression of the Neurite Growth-Associated Protein B-50/GAP-43 in Cerebellar Purkinje Cells Induces Sprouting after Axotomy But Not Axon Regeneration into Growth-Permissive TransplantsJournal of Neuroscience, 1997
- A Nonimmunosuppressant FKBP-12 Ligand Increases Nerve RegenerationExperimental Neurology, 1997
- Adenoviral Vector-Mediated Expression of B-50/GAP-43 Induces Alterations in the Membrane Organization of Olfactory Axon TerminalsIn VivoJournal of Neuroscience, 1997
- GAP-43: an intrinsic determinant of neuronal development and plasticityTrends in Neurosciences, 1997
- The immunosuppressant FK506 increases the rate of axonal regeneration in rat sciatic nerveJournal of Neuroscience, 1995
- Overexpression of the neural growth-associated protein GAP-43 induces nerve sprouting in the adult nervous system of transgenic miceCell, 1995
- The immunosuppressive and toxic effects of FK-506 are mechanistically related: pharmacology of a novel antagonist of FK-506 and rapamycin.The Journal of Experimental Medicine, 1992
- Factors influencing GAP-43 gene expression in PC12 pheochromocytoma cellsJournal of Neuroscience, 1990
- Environmental influences in the development of neural crest derivatives: glucocorticoids, growth factors, and chromaffin cell plasticityJournal of Neuroscience, 1985