Degenerative Disorders Caused by Bcl-2 Deficiency Prevented by Loss of Its BH3-Only Antagonist Bim
- 30 November 2001
- journal article
- Published by Elsevier BV in Developmental Cell
- Vol. 1 (5), 645-653
- https://doi.org/10.1016/s1534-5807(01)00083-1
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and BakGenes & Development, 2001
- Translocation of C. elegans CED-4 to Nuclear Membranes During Programmed Cell DeathScience, 2000
- Bcl-2 inhibits Bax translocation from cytosol to mitochondria during drug-induced apoptosis of human tumor cellsCell Death & Differentiation, 2000
- Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude AutoimmunityScience, 1999
- Bcl-2 overexpression prevents apoptosis-induced Madin-Darby canine kidney simple epithelial cyst formationKidney International, 1999
- The Bcl-2 Protein Family: Arbiters of Cell SurvivalScience, 1998
- Bcl-2 Rescues T Lymphopoiesis in Interleukin-7 Receptor–Deficient MiceCell, 1997
- Bcl-2 Can Rescue T Lymphocyte Development in Interleukin-7 Receptor–Deficient Mice but Not in Mutant rag-1−/− MiceCell, 1997
- Acre-transgenic mouse strain for the ubiquitous deletion ofloxP-flanked gene segments including deletion in germ cellsNucleic Acids Research, 1995
- Bcl-2-deficient mice demonstrate fulminant lymphoid apoptosis, polycystic kidneys, and hypopigmented hairCell, 1993