The p38 mitogen-activated protein kinase pathway—A potential target for intervention in infarction, hypertrophy, and heart failure
- 31 October 2011
- journal article
- review article
- Published by Elsevier BV in Journal of Molecular and Cellular Cardiology
- Vol. 51 (4), 485-490
- https://doi.org/10.1016/j.yjmcc.2010.10.021
Abstract
No abstract availableKeywords
This publication has 84 references indexed in Scilit:
- The activation of p38alpha, and not p38beta, mitogen-activated protein kinase is required for ischemic preconditioningJournal of Molecular and Cellular Cardiology, 2010
- Amyloidogenic light chains induce cardiomyocyte contractile dysfunction and apoptosis via a non-canonical p38α MAPK pathwayProceedings of the National Academy of Sciences of the United States of America, 2010
- A Chemical Genetic Approach Reveals That p38α MAPK Activation by Diphosphorylation Aggravates Myocardial Infarction and Is Prevented by the Direct Binding of SB203580Published by Elsevier BV ,2010
- Sustained release of a p38 inhibitor from non-inflammatory microspheres inhibits cardiac dysfunctionNature Materials, 2008
- A helix scaffold for the assembly of active protein kinasesProceedings of the National Academy of Sciences of the United States of America, 2008
- The selectivity of protein kinase inhibitors: a further updateBiochemical Journal, 2007
- p38γ regulates the localisation of SAP97 in the cytoskeleton by modulating its interaction with GKAPThe EMBO Journal, 2005
- Disruption of a single copy of the p38α MAP kinase gene leads to cardioprotection against ischemia–reperfusionBiochemical and Biophysical Research Communications, 2003
- Activation of p38 MAPK Induced by a Multi-cycle Ischaemic Preconditioning Protocol is Associated with Attenuated p38 MAPK Activity During Sustained Ischaemia and ReperfusionJournal of Molecular and Cellular Cardiology, 2001
- Activation of c-Jun N-Terminal Kinases and p38-Mitogen-activated Protein Kinases in Human Heart Failure Secondary to Ischaemic Heart DiseaseJournal of Molecular and Cellular Cardiology, 1999