Calpain and Mitochondria in Ischemia/Reperfusion Injury
Open Access
- 1 August 2002
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 277 (32), 29181-29186
- https://doi.org/10.1074/jbc.m204951200
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Role of the Mitochondrial Permeability Transition and Cytochrome c Release in Hydrogen Peroxide-Induced ApoptosisExperimental Cell Research, 2002
- Endonuclease G: a mitochondrial protein released in apoptosis and involved in caspase-independent DNA degradationCell Death & Differentiation, 2001
- Therapeutic potential of Na-H exchange inhibitors for the treatment of heart failureExpert Opinion on Investigational Drugs, 2001
- Pharmacology of Caspase Inhibitors in Rabbit Cardiomyocytes Subjected to Metabolic Inhibition and RecoveryAntioxidants and Redox Signaling, 2001
- Smac, a Mitochondrial Protein that Promotes Cytochrome c–Dependent Caspase Activation by Eliminating IAP InhibitionCell, 2000
- Co-localization of the Cysteine Protease Caspase-3 with Apoptotic Myocytes afterIn VivoMyocardial Ischemia and Reperfusion in the RatJournal of Molecular and Cellular Cardiology, 1998
- MDL-28170, a membrane-permeant calpain inhibitor, attenuates stunning and PKCε proteolysis in reperfused ferret heartsCardiovascular Research, 1997
- Preconditioning rabbit cardiomyocytes: role of pH, vacuolar proton ATPase, and apoptosis.JCI Insight, 1996
- Reperfusion injury induces apoptosis in rabbit cardiomyocytes.JCI Insight, 1994
- Simvastatin‐sodium delays cell death of anoxic cardiomyocytes by inhibition of the Na+/Ca2+ exchangerFEBS Letters, 1994