The mode of cisplatin-induced cell death in CYP2E1-overexpressing HepG2 cells: Modulation by ERK, ROS, glutathione, and thioredoxin
- 1 October 2007
- journal article
- Published by Elsevier BV in Free Radical Biology & Medicine
- Vol. 43 (7), 1061-1075
- https://doi.org/10.1016/j.freeradbiomed.2007.06.021
Abstract
No abstract availableKeywords
This publication has 71 references indexed in Scilit:
- Caspases and calpain are independent mediators of cisplatin-induced endothelial cell necrosisAmerican Journal of Physiology-Renal Physiology, 2006
- A Death-Promoting Role for Extracellular Signal-Regulated KinaseJournal of Pharmacology and Experimental Therapeutics, 2006
- Cisplatin-Induced Hepatotoxicity Is Enhanced by Elevated Expression of Cytochrome P450 2E1Toxicological Sciences, 2005
- Lipopolysaccharide-induced liver injury in rats treated with the CYP2E1 inducer pyrazoleAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2005
- Cisplatin Inactivation of Caspases Inhibits Death Ligand-induced Cell Death in Vitro and Fulminant Liver Damage in MicePublished by Elsevier BV ,2005
- A Protein Interaction Map of Drosophila melanogasterScience, 2003
- Recent Trends in Glutathione Biochemistry—Glutathione–Protein Interactions: A Molecular Link between Oxidative Stress and Cell Proliferation?Biochemical and Biophysical Research Communications, 1998
- Serrate and wingless cooperate to induce vestigial gene expression and wing formation in DrosophilaCurrent Biology, 1995
- Cisplatin Generates Superoxide Anion by Interaction with DNA in a Cell-Free SystemBiochemical and Biophysical Research Communications, 1994
- Increased release of hydrogen peroxide (H2O2) and superoxide anion (O2−) by murine macrophages in vitro after cis-Platin treatmentInternational Journal of Immunopharmacology, 1986