Inhibition of the vanilloid receptor subtype-1 attenuates TNBS-colitis
- 1 November 2004
- journal article
- Published by Elsevier BV in Journal of Gastrointestinal Surgery
- Vol. 8 (7), 842-848
- https://doi.org/10.1016/j.gassur.2004.07.011
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- An interleukin 12 p40-IgG2b fusion protein abrogates T cell mediated inflammation: anti-inflammatory activity in Crohn's disease and experimental colitis in vivoGut, 2004
- Substance P induces tumor necrosis factor-α release from human skin via mitogen-activated protein kinaseEuropean Journal of Pharmacology, 2000
- Neurokinin-1 (NK-1) Receptor Is Required in Antigen-Induced CystitisThe American Journal of Pathology, 2000
- The effect of an inhibitor of matrix metalloproteinases on colonic inflammation in a trinitrobenzenesulphonic acid rat model of inflammatory bowel diseaseAlimentary Pharmacology & Therapeutics, 1999
- Predominant pathogenic role of tumor necrosis factor in experimental colitis in miceEuropean Journal of Immunology, 1997
- Substance P activation of enteric neurons in response to intraluminal Clostridium difficile toxin A in the rat ileumGastroenterology, 1996
- Induction of tachykinin gene and peptide expression in guinea pig nodose primary afferent neurons by allergic airway inflammation.JCI Insight, 1996
- CP-96,345, a substance P antagonist, inhibits rat intestinal responses to Clostridium difficile toxin A but not cholera toxin.Proceedings of the National Academy of Sciences, 1994
- Capsazepine: a competitive antagonist of the sensory neurone excitant capsaicinBritish Journal of Pharmacology, 1992
- Receptor binding sites for substance P, but not substance K or neuromedin K, are expressed in high concentrations by arterioles, venules, and lymph nodules in surgical specimens obtained from patients with ulcerative colitis and Crohn disease.Proceedings of the National Academy of Sciences, 1988