Integration of the full-length HPV16 genome in cervical cancer and Caski and Siha cell lines and the possible ways of HPV integration
- 10 February 2015
- journal article
- research article
- Published by Springer Science and Business Media LLC in Virus Genes
- Vol. 50 (2), 210-220
- https://doi.org/10.1007/s11262-014-1164-7
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Multiplex Identification of Human Papillomavirus 16 DNA Integration Sites in Cervical CarcinomasPLOS ONE, 2013
- Analysis of Human Papillomavirus Type 16 (HPV16) DNA Load and Physical State for Identification of HPV16-Infected Women with High-Grade Lesions or Cervical CarcinomaJournal of Clinical Microbiology, 2008
- Viral DNA load, physical status and E2/E6 ratio as markers to grade HPV16 positive women for high-grade cervical lesionsGynecologic Oncology, 2007
- Integration of high‐risk human papillomavirus: a key event in cervical carcinogenesis?The Journal of Pathology, 2007
- Human papillomavirus status in advanced cervical cancer: predictive and prognostic significance for curative radiation treatmentInternational Journal of Gynecologic Cancer, 2005
- Mechanisms of Human Papillomavirus-Induced OncogenesisJournal of Virology, 2004
- Clonal Selection for Transcriptionally Active Viral Oncogenes during Progression to CancerJournal of Virology, 2004
- Episomal and integrated human papillomavirus in cervical neoplasia shown by non-isotopic in situ hybridisation.Journal of Clinical Pathology, 1991
- Physical State and Biological Activity of Human Papillomavirus Genomes in Precancerous Lesions of the Female Genital TractJournal of General Virology, 1988
- The Physical State of Human Papillomavirus Type 16 DNA in Benign and Malignant Genital TumoursJournal of General Virology, 1985