Pot1 Deficiency Initiates DNA Damage Checkpoint Activation and Aberrant Homologous Recombination at Telomeres
- 1 July 2006
- journal article
- research article
- Published by Elsevier BV in Cell
- Vol. 126 (1), 49-62
- https://doi.org/10.1016/j.cell.2006.05.037
Abstract
No abstract availableThis publication has 63 references indexed in Scilit:
- Frequent recombination in telomeric DNA may extend the proliferative life of telomerase-negative cellsNucleic Acids Research, 2004
- Telomeric Recombination in Mismatch Repair Deficient Human Colon Cancer Cells after Telomerase InhibitionCancer Research, 2004
- Human Pot1 (Protection of Telomeres) Protein: Cytolocalization, Gene Structure, and Alternative SplicingMolecular and Cellular Biology, 2002
- Strand-Specific Postreplicative Processing of Mammalian TelomeresScience, 2001
- Switching and Signaling at the TelomereCell, 2001
- Pot1, the Putative Telomere End-Binding Protein in Fission Yeast and HumansScience, 2001
- Telomere dysfunction promotes non-reciprocal translocations and epithelial cancers in miceNature, 2000
- Targeted Mutagenesis of the Endogenous Mouse Mis Gene PromoterCell, 1999
- Enhanced Phosphorylation of p53 by ATM in Response to DNA DamageScience, 1998
- Telomere Shortening and Tumor Formation by Mouse Cells Lacking Telomerase RNACell, 1997