Apoptosis, transforming growth factor‐β, and the immunosuppressive effect of transfusion
- 16 September 2002
- journal article
- Published by Wiley in Transfusion
- Vol. 42 (9), 1221-1223
- https://doi.org/10.1046/j.1537-2995.2002.00175.x
Abstract
No abstract availableThis publication has 17 references indexed in Scilit:
- Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-β1 secretion and the resolution of inflammationJCI Insight, 2002
- Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-β1 secretion and the resolution of inflammationJCI Insight, 2002
- Intravenous injection of apoptotic leukocytes enhances bone marrow engraftment across major histocompatibility barriersBlood, 2001
- A receptor for phosphatidylserine-specific clearance of apoptotic cellsNature, 2000
- REGULATION OF IMMUNE RESPONSES BY TGF-βAnnual Review of Immunology, 1998
- Beneficial Effects of Leukocyte Depletion of Transfused Blood on Postoperative Complications in Patients Undergoing Cardiac SurgeryCirculation, 1998
- Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF.JCI Insight, 1998
- Transforming growth factor beta is increased in plasma of patients with hematologic malignancies after transfusion of platelet concentratesTransfusion, 1998
- Control of nitric oxide synthase expression by transforming growth factor-β: Implications for homeostasisProgress in Growth Factor Research, 1994
- Pretransplantation Blood Transfusion RevisitedThe New England Journal of Medicine, 1991