Th1 Immune Response Promotes Severe Bone Resorption Caused by Porphyromonas gingivalis
- 31 January 2007
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 170 (1), 203-213
- https://doi.org/10.2353/ajpath.2007.060597
Abstract
No abstract availableKeywords
This publication has 72 references indexed in Scilit:
- Interleukin-10 Inhibits Gram-Negative-Microbe-Specific Human Receptor Activator of NF-κB Ligand-Positive CD4 + -Th1-Cell- Associated Alveolar Bone Loss In VivoInfection and Immunity, 2006
- T-Cell Expression Cloning ofPorphyromonas gingivalisGenes Coding for T Helper-Biased Immune Responses during InfectionInfection and Immunity, 2006
- Periodontal and Hematological Characteristics Associated With Aggressive Periodontitis, Juvenile Idiopathic Arthritis, and Rheumatoid ArthritisThe Journal of Periodontology, 2006
- IL-1α stimulates the formation of osteoclast-like cells by increasing M-CSF and PGE2 production and decreasing OPG production by osteoblastsLife Sciences, 2005
- IL-6, RANKL, TNF-alpha/IL-1: interrelations in bone resorption pathophysiologyCytokine & Growth Factor Reviews, 2004
- Interleukin (Il)-18 Promotes the Development of Chronic Gastrointestinal Helminth Infection by Downregulating IL-13The Journal of Experimental Medicine, 2001
- Relationship Between Rheumatoid Arthritis and PeriodontitisThe Journal of Periodontology, 2001
- Conversion in vivo from an early dominant Th0/Th1 response to a Th2 phenotype during the development of collagen‐induced arthritisEuropean Journal of Immunology, 1997
- Estrogen Stimulates the Elaboration of Cell/Matrix Surface-Associated Inhibitory Factor of Osteoclastic Bone Resorption from Osteoblastic CellsBiochemical and Biophysical Research Communications, 1993
- The Bacterial Etiology of Destructive Periodontal Disease: Current ConceptsThe Journal of Periodontology, 1992