Adenomatous Polyposis Coli (APC)-independent Regulation of β-Catenin Degradation via a Retinoid X Receptor-mediated Pathway
Open Access
- 1 August 2003
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 278 (32), 29954-29962
- https://doi.org/10.1074/jbc.m304761200
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Nr-CAM is a target gene of the β-catenin/LEF-1 pathway in melanoma and colon cancer and its expression enhances motility and confers tumorigenesisGenes & Development, 2002
- Activation of the β-Catenin/Lef-Tcf Pathway Is Obligate for Formation of Primitive Endoderm by Mouse F9 Totipotent Teratocarcinoma Cells in Response to Retinoic AcidPublished by Elsevier BV ,2002
- Synergistic Induction of Tumor Antigens by Wnt-1 Signaling and Retinoic Acid Revealed by Gene Expression ProfilingJournal of Biological Chemistry, 2002
- Early development of polycystic kidney disease in transgenic mice expressing an activated mutant of the β-catenin geneOncogene, 2001
- Dimerization with Retinoid X Receptors and Phosphorylation Modulate the Retinoic Acid-induced Degradation of Retinoic Acid Receptors α and γ through the Ubiquitin-Proteasome PathwayPublished by Elsevier BV ,2000
- Wnt Signaling in Oncogenesis and Embryogenesis--a Look Outside the NucleusScience, 2000
- Cross-regulation of β-catenin–LEF/TCF and retinoid signaling pathwaysCurrent Biology, 1999
- Intestinal polyposis in mice with a dominant stable mutation of the beta -catenin geneThe EMBO Journal, 1999
- Constitutive Transcriptional Activation by a β-Catenin-Tcf Complex in APC −/− Colon CarcinomaScience, 1997
- Estrogen Induces Retinoid Receptor Expression in Mouse Cervical EpitheliaExperimental Cell Research, 1996