How interleukin‐1β induces gouty arthritis
Open Access
- 29 October 2010
- journal article
- editorial
- Published by Wiley in Arthritis & Rheumatism
- Vol. 62 (11), 3140-3144
- https://doi.org/10.1002/art.27663
Abstract
No abstract availableThis publication has 16 references indexed in Scilit:
- Engagement of fatty acids with toll‐like receptor 2 drives interleukin‐1β production via the ASC/caspase 1 pathway in monosodium urate monohydrate crystal–induced gouty arthritisArthritis & Rheumatism, 2010
- Inflammatory arthritis in caspase 1 gene–deficient mice: Contribution of proteinase 3 to caspase 1–independent production of bioactive interleukin‐1βArthritis & Rheumatism, 2009
- Inflammatory Role of ASC in Antigen-Induced Arthritis Is Independent of Caspase-1, NALP-3, and IPAFThe Journal of Immunology, 2009
- The interleukin 1 inhibitor rilonacept in treatment of chronic gouty arthritis: results of a placebo-controlled, monosequence crossover, non-randomised, single-blind pilot studyAnnals Of The Rheumatic Diseases, 2009
- Differential requirement for the activation of the inflammasome for processing and release of IL-1β in monocytes and macrophagesBlood, 2009
- Crystals of monosodium urate monohydrate enhance lipopolysaccharide-induced release of interleukin 1β by mononuclear cells through a caspase 1-mediated processAnnals Of The Rheumatic Diseases, 2008
- A Subpopulation of Macrophages Infiltrates Hypertrophic Adipose Tissue and Is Activated by Free Fatty Acids via Toll-like Receptors 2 and 4 and JNK-dependent PathwaysPublished by Elsevier BV ,2007
- MyD88-dependent IL-1 receptor signaling is essential for gouty inflammation stimulated by monosodium urate crystalsJCI Insight, 2006
- Gout-associated uric acid crystals activate the NALP3 inflammasomeNature, 2006
- Endothelial activation in monosodium urate monohydrate crystal‐induced inflammation. In vitro and in vivo studies on the roles of tumor necrosis factor α and interleukin‐1Arthritis & Rheumatism, 1997