NF1 Is a Tumor Suppressor in Neuroblastoma that Determines Retinoic Acid Response and Disease Outcome
- 23 July 2010
- journal article
- Published by Elsevier BV in Cell
- Vol. 142 (2), 218-229
- https://doi.org/10.1016/j.cell.2010.06.004
Abstract
No abstract availableThis publication has 48 references indexed in Scilit:
- Proteasomal and Genetic Inactivation of the NF1 Tumor Suppressor in GliomagenesisCancer Cell, 2009
- ZNF423 Is Critically Required for Retinoic Acid-Induced Differentiation and Is a Marker of Neuroblastoma OutcomeCancer Cell, 2009
- Long-Term Results for Children With High-Risk Neuroblastoma Treated on a Randomized Trial of Myeloablative Therapy Followed by 13-cis-Retinoic Acid: A Children's Oncology Group StudyJournal of Clinical Oncology, 2009
- A coordinated phosphorylation cascade initiated by p38MAPK/MSK1 directs RARα to target promotersThe EMBO Journal, 2008
- K-rasMutations and Benefit from Cetuximab in Advanced Colorectal CancerThe New England Journal of Medicine, 2008
- Retinoic Acid Synthesis and Signaling during Early OrganogenesisCell, 2008
- Comprehensive genomic characterization defines human glioblastoma genes and core pathwaysNature, 2008
- Activated Kras, but Not Hras or Nras, May Initiate Tumors of Endodermal Origin via Stem Cell ExpansionMolecular and Cellular Biology, 2008
- A functional genetic screen identifies retinoic acid signaling as a target of histone deacetylase inhibitorsProceedings of the National Academy of Sciences of the United States of America, 2007
- Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescenceNature, 2006