The impact of genomic imprinting for neurobehavioral and developmental disorders
Open Access
- 15 February 2000
- journal article
- review article
- Published by American Society for Clinical Investigation in JCI Insight
- Vol. 105 (4), 413-418
- https://doi.org/10.1172/jci9460
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Incriminating gene suspects, Prader-Willi styleNature Genetics, 1999
- A maternally methylated CpG island in KvLQT1 is associated with an antisense paternal transcript and loss of imprinting in Beckwith–Wiedemann syndromeProceedings of the National Academy of Sciences of the United States of America, 1999
- Genetics of Angelman SyndromeAmerican Journal of Human Genetics, 1999
- Loss of imprinting of a paternally expressed transcript, with antisense orientation to K V LQT1, occurs frequently in Beckwith–Wiedemann syndrome and is independent of insulin-like growth factor II imprintingProceedings of the National Academy of Sciences of the United States of America, 1999
- Regulation of Maternal Behavior and Offspring Growth by Paternally Expressed Peg3Science, 1999
- A cluster of oppositely imprinted transcripts at the Gnas locus in the distal imprinting region of mouse chromosome 2Proceedings of the National Academy of Sciences of the United States of America, 1999
- Evaluation of linkage of bipolar affective disorder to chromosome 18 in a sample of 57 German familiesMolecular Psychiatry, 1999
- Bidirectional imprinting of a single gene: GNAS1 encodes maternally, paternally, and biallelically derived proteinsProceedings of the National Academy of Sciences of the United States of America, 1998
- Patterns of maternal transmission in bipolar affective disorder.1995
- Association of Decreased Paternal Age and Late-Onset Alzheimer's DiseaseArchives of Neurology, 1991