Targeting of the 67-kDa Isoform of Glutamic Acid Decarboxylase to Intracellular Organelles Is Mediated by Its Interaction with the NH2-terminal Region of the 65-kDa Isoform of Glutamic Acid Decarboxylase
Open Access
- 1 February 1995
- journal article
- Published by Elsevier BV in Online Journal of Public Health Informatics
- Vol. 270 (5), 2241-2246
- https://doi.org/10.1074/jbc.270.5.2241
Abstract
No abstract availableThis publication has 34 references indexed in Scilit:
- Formation of synaptic vesiclesCurrent Opinion in Cell Biology, 1994
- Demonstration of GAD-65 as the main immunogenic isoform of glutamate decarboxylase in type 1 diabetes and determination of autoantibodies using a radioligand produced by eukaryotic expression.JCI Insight, 1993
- Colocalization of synaptophysin with transferrin receptors: implications for synaptic vesicle biogenesis.The Journal of cell biology, 1991
- Autoantibodies to GABA-ergic Neurons and Pancreatic Beta Cells in Stiff-Man SyndromeThe New England Journal of Medicine, 1990
- Amino acid neurotransmission: spotlight on synaptic vesiclesTrends in Neurosciences, 1990
- Expression of a multifunctional Ca2+/calmodulin-dependent protein kinase and mutational analysis of its autoregulationNeuron, 1989
- Autoantibodies to Glutamic Acid Decarboxylase in a Patient with Stiff-Man Syndrome, Epilepsy, and Type I Diabetes MellitusThe New England Journal of Medicine, 1988
- Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications.Proceedings of the National Academy of Sciences of the United States of America, 1979
- The fine structural localization of glutamate decarboxylase in developing axonal processes and presynaptic terminals of rodent cerebellumBrain Research, 1975
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970