Temporally Regulated Neural Crest Transcription Factors Distinguish Neuroectodermal Tumors of Varying Malignancy and Differentiation
Open Access
- 1 June 2005
- journal article
- Published by Elsevier BV in Neoplasia
- Vol. 7 (6), 575-584
- https://doi.org/10.1593/neo.04637
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Conserved mechanisms across development and tumorigenesis revealed by a mouse development perspective of human cancersGenes & Development, 2004
- Notch and Schwann cell transformationOncogene, 2004
- A Subtractive Gene Expression Screen Suggests a Role of Transcription Factor AP-2α in Control of Proliferation and DifferentiationJournal of Biological Chemistry, 2002
- Prediction of central nervous system embryonal tumour outcome based on gene expressionNature, 2002
- Dominant-negative transcription factor AP-2 augments SB-2 melanoma tumor growth in vivoOncogene, 2001
- Overexpression of Murine Pax3 Increases NCAM Polysialylation in a Human Medulloblastoma Cell LineJournal of Biological Chemistry, 2000
- Immunohistochemical analysis reveals a tumour suppressor-like role for the transcription factor AP-2 in invasive breast cancerThe Journal of Pathology, 1999
- SOX10 mutation disrupts neural crest development in Dom Hirschsprung mouse modelNature Genetics, 1998
- AP2 inhibits cancer cell growth and activates p21WAF1/CIP1 expressionNature Genetics, 1997
- Transcription factor AP-2 essential for cranial closure and craniofacial developmentNature, 1996