The X11α Protein Slows Cellular Amyloid Precursor Protein Processing and Reduces Aβ40 and Aβ42 Secretion
Open Access
- 1 June 1998
- journal article
- Published by Elsevier BV
- Vol. 273 (24), 14761-14766
- https://doi.org/10.1074/jbc.273.24.14761
Abstract
No abstract availableThis publication has 36 references indexed in Scilit:
- Novel β-Secretase Cleavage of β-Amyloid Precursor Protein in the Endoplasmic Reticulum/Intermediate Compartment of NT2N CellsThe Journal of cell biology, 1997
- Interaction of the Phosphotyrosine Interaction/Phosphotyrosine Binding-related Domains of Fe65 with Wild-type and Mutant Alzheimer's β-Amyloid Precursor ProteinsPublished by Elsevier BV ,1997
- The intracellular cytoplasmic domain of the Alzheimer's disease amyloid precursor protein interacts with phosphotyrosine‐binding domain proteins in the yeast two‐hybrid systemFEBS Letters, 1996
- Control of Daughter Cell Fates during Asymmetric Division: Interaction of Numb and NotchNeuron, 1996
- Amyloids and Are Generated Intracellularly in Cultured Human Neurons and Their Secretion Increases with MaturationPublished by Elsevier BV ,1996
- The Regions of the Fe65 Protein Homologous to the Phosphotyrosine Interaction/Phosphotyrosine Binding Domain of Shc Bind the Intracellular Domain of the Alzheimer's Amyloid Precursor ProteinPublished by Elsevier BV ,1995
- Intracellular Accumulation of β-Amyloid in Cells Expressing the Swedish Mutant Amyloid Precursor ProteinPublished by Elsevier BV ,1995
- An Alternative to SH2 Domains for Binding Tyrosine-Phosphorylated ProteinsScience, 1994
- Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein PrecursorScience, 1993
- Mutation of the β-amyloid precursor protein in familial Alzheimer's disease increases β-protein productionNature, 1992