CK2 Is a C-Terminal IκB Kinase Responsible for NF-κB Activation during the UV Response
Open Access
- 31 October 2003
- journal article
- research article
- Published by Elsevier BV in Molecular Cell
- Vol. 12 (4), 829-839
- https://doi.org/10.1016/s1097-2765(03)00358-7
Abstract
No abstract availableFunding Information
- American Cancer Society
- Ministry of Education, Culture, Sports, Science and Technology
- National Institutes of Health (CA76188)
This publication has 41 references indexed in Scilit:
- p53 stabilization is decreased upon NFκB activation: A role for NFκB in acquisition of resistance to chemotherapyCancer Cell, 2002
- NF-κB at the crossroads of life and deathNature Immunology, 2002
- Requirement for p38α in Erythropoietin Expression: A Role for Stress Kinases in ErythropoiesisCell, 2000
- Signal-dependent and -independent Degradation of Free and NF-κB-bound IκBαOnline Journal of Public Health Informatics, 2000
- Stress-induced Activation of Protein Kinase CK2 by Direct Interaction with p38 Mitogen-activated Protein KinaseOnline Journal of Public Health Informatics, 2000
- A Subclass of Ras Proteins That Regulate the Degradation of IκBScience, 2000
- Positive and Negative Regulation of IκB Kinase Activity Through IKKβ Subunit PhosphorylationScience, 1999
- NF-κB AND REL PROTEINS: Evolutionarily Conserved Mediators of Immune ResponsesAnnual Review of Immunology, 1998
- The IκB Kinase Complex (IKK) Contains Two Kinase Subunits, IKKα and IKKβ, Necessary for IκB Phosphorylation and NF-κB ActivationCell, 1997
- Expression of the cDNA for the beta subunit of human casein kinase II confers partial UV resistance on xeroderma pigmentosum cellsMutation Research/DNA Repair, 1990