The ubiquitin–proteasome system postsynaptically regulates glutamatergic synaptic function
- 31 May 2007
- journal article
- Published by Elsevier BV in Molecular and Cellular Neuroscience
- Vol. 35 (1), 64-75
- https://doi.org/10.1016/j.mcn.2007.02.002
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (NS048882-01, NS41740)
This publication has 64 references indexed in Scilit:
- Mechanisms Underlying the Rapid Induction and Sustained Expression of Synaptic HomeostasisNeuron, 2006
- A Genetic Suppressor of Two Dominant Temperature-Sensitive Lethal Proteasome Mutants of Drosophila melanogaster Is Itself a Mutated Proteasome Subunit GeneGenetics, 2006
- KEL-8 Is a Substrate Receptor for CUL3-dependent Ubiquitin Ligase That Regulates Synaptic Glutamate Receptor TurnoverMolecular Biology of the Cell, 2006
- Involvement of a cellular ubiquitin-protein ligase E6AP in the ubiquitin-mediated degradation of extensive substrates of high-risk human papillomavirus E6Journal of Medical Virology, 2006
- Ubiquitin and Protein Turnover in Synapse FunctionNeuron, 2005
- Independent Regulation of Synaptic Size and Activity by the Anaphase-Promoting ComplexCell, 2004
- The Anaphase-Promoting Complex Regulates the Abundance of GLR-1 Glutamate Receptors in the Ventral Nerve Cord of C. elegansCurrent Biology, 2004
- Targeted expression of dominant negative proteasome mutants indrosophila melanogastergenesis, 2002
- Recognition of the polyubiquitin proteolytic signalThe EMBO Journal, 2000
- Syntaxin and synaptobrevin function downstream of vesicle docking in drosophilaNeuron, 1995