Sustained potentiation of NMDA receptor-mediated glutamate responses through activation of protein kinase C by a μ opioid
- 31 August 1991
- journal article
- Published by Elsevier BV in Neuron
- Vol. 7 (2), 319-326
- https://doi.org/10.1016/0896-6273(91)90270-a
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Activation of μ opioid receptors inhibits transient high- and low-threshold Ca 2+ currents, but spares a sustained currentNeuron, 1991
- Molecular Characterization of Opioid ReceptorsAnnual Review of Pharmacology and Toxicology, 1990
- Specific inhibitors of protein kinase C block transmitter-induced modulation of sensory neuron calcium currentNeuron, 1989
- Anatomy of CNS opioid receptorsTrends in Neurosciences, 1988
- Opioid peptide gene expression in rat trigeminal nucleus caudalis neurons: Normal distribution and effects of trigeminal deafferentationJournal of Comparative Neurology, 1988
- Increased staining of immunoreactive dynorphin cell bodies in the deafferented spinal cord of the ratNeuroscience Letters, 1988
- Selective excitation of neurons in the mammalian spinal dorsal horn by aspartate and glutamate in vitro: correlation with location and excitatory inputBrain Research, 1985
- The Physiology and Pharmacology of Spinal OpiatesAnnual Review of Pharmacology and Toxicology, 1985
- Magnesium gates glutamate-activated channels in mouse central neuronesNature, 1984
- Chronic peripheral nerve section diminishes the primary afferent A-fibre mediated inhibition of rat dorsal horn neuronesBrain Research, 1982