Hyperresponsiveness of neutrophils from gp 91phox deficient patients to lipopolysaccharide and serum amyloid A
- 15 June 2004
- journal article
- research article
- Published by Elsevier BV in Immunology Letters
- Vol. 94 (1-2), 43-46
- https://doi.org/10.1016/j.imlet.2004.04.016
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Serum amyloid A-induced mRNA expression and release of tumor necrosis factor-alpha (TNF-α) in human neutrophilsImmunology Letters, 2003
- RELATIVE CYTOKINE AND CYTOKINE INHIBITOR PRODUCTION BY MONONUCLEAR CELLS AND NEUTROPHILSShock, 2003
- Holding back neutrophil aggression; the oxidase has potentialClinical and Experimental Immunology, 2003
- Calcium signalling is altered in myeloid cells with a deficiency in NADPH oxidase activityClinical and Experimental Immunology, 2003
- Accelerated calcium influx and hyperactivation of neutrophils in chronic granulomatous diseaseClinical and Experimental Immunology, 2001
- Chronic granulomatous diseaseClinical and Experimental Immunology, 2000
- EXPRESSION OF IL-8 GENE IN HUMAN MONOCYTES AND LYMPHOCYTES: DIFFERENTIAL REGULATION BY TNF AND IL-1Cytokine, 2000
- A Novel Function of Serum Amyloid A: A Potent Stimulus for the Release of Tumor Necrosis Factor-α, Interleukin-1β, and Interleukin-8 by Human Blood NeutrophilBiochemical and Biophysical Research Communications, 2000
- mRNA expression and release of interleukin‐8 induced by serum amyloid A in neutrophils and monocytesMediators of Inflammation, 2000
- Human Chemokines: An UpdateAnnual Review of Immunology, 1997