Extracellular Nampt Promotes Macrophage Survival via a Nonenzymatic Interleukin-6/STAT3 Signaling Mechanism
Open Access
- 1 December 2008
- journal article
- Published by Elsevier BV
- Vol. 283 (50), 34833-34843
- https://doi.org/10.1074/jbc.m805866200
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- Increase in Endoplasmic Reticulum Stress–Related Proteins and Genes in Adipose Tissue of Obese, Insulin-Resistant IndividualsDiabetes, 2008
- Pharmacological Inhibition of Nicotinamide Phosphoribosyltransferase/Visfatin Enzymatic Activity Identifies a New Inflammatory Pathway Linked to NADPLOS ONE, 2008
- Anticancer agent CHS-828 inhibits cellular synthesis of NADBiochemical and Biophysical Research Communications, 2008
- The wolf in sheep's clothing: the role of interleukin-6 in immunity, inflammation and cancerTrends in Molecular Medicine, 2008
- Nampt/PBEF/Visfatin Regulates Insulin Secretion in β Cells as a Systemic NAD Biosynthetic EnzymeCell Metabolism, 2007
- Increased Expression of Visfatin in Macrophages of Human Unstable Carotid and Coronary AtherosclerosisCirculation, 2007
- Combinatorial pattern recognition receptor signaling alters the balance of life and death in macrophagesProceedings of the National Academy of Sciences of the United States of America, 2006
- The role of IL-6 and STAT3 in inflammation and cancerEuropean Journal Of Cancer, 2005
- Tuning of macrophage responses by Stat3-inducing cytokines: molecular mechanisms and consequences in infectionImmunobiology, 2005
- The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophagesNature, 2003