Methylation of the p16Ink4a tumor suppressor gene 5′-CpG island in breast cancer
- 10 February 2001
- journal article
- research article
- Published by Elsevier BV in Cancer Letters
- Vol. 163 (1), 59-69
- https://doi.org/10.1016/s0304-3835(00)00674-1
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Methylated DNA and MeCP2 recruit histone deacetylase to repress transcriptionNature Genetics, 1998
- Transcriptional repression by the methyl-CpG-binding protein MeCP2 involves a histone deacetylase complexNature, 1998
- Retinoblastoma and p16 proteins in mammary carcinoma: Their relationship to cyclin D1 and histopathological parametersInternational Journal of Cancer, 1998
- Infrequent CDKN2 (MTS1/p16) gene alterations in human primary breast cancerBritish Journal of Cancer, 1995
- Frequency of homozygous deletion at p16/CDKN2 in primary human tumoursNature Genetics, 1995
- Analysis of p16 gene deletion and point mutation in breast carcinomaBritish Journal of Cancer, 1995
- 5′ CpG island methylation is associated with transcriptional silencing of the tumour suppressor p16/CDKN2/MTS1 in human cancersNature Medicine, 1995
- Analysis of MTS1/CDK4 in female breast carcinomasCancer Letters, 1995
- Sp1 sites in the mouse aprt gene promoter are required to prevent methylation of the CpG island.Genes & Development, 1994
- Spl elements protect a CpG island from de novo methylationNature, 1994