γ-Catenin expression is reduced or absent in a subset of human lung cancers and re-expression inhibits transformed cell growth
- 18 October 2002
- journal article
- Published by Springer Science and Business Media LLC in Oncogene
- Vol. 21 (49), 7497-7506
- https://doi.org/10.1038/sj.onc.1205963
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 36 references indexed in Scilit:
- The Transcriptional Factor Tcf-4 Contains Different Binding Sites for β-Catenin and PlakoglobinJournal of Biological Chemistry, 2002
- EGFR and cancer prognosisEuropean Journal Of Cancer, 2001
- Cancer Statistics, 2001CA: A Cancer Journal for Clinicians, 2001
- Co-expression of epidermal growth factor receptor and transforming growth factor-α is independent of ras mutations in lung adenocarcinomaLung Cancer, 2000
- Differential Mechanisms of LEF/TCF Family-Dependent Transcriptional Activation by β-Catenin and PlakoglobinMolecular and Cellular Biology, 2000
- The metalloproteinase matrilysin is a target of β-catenin transactivation in intestinal tumorsOncogene, 1999
- Activation of β-Catenin-Tcf Signaling in Colon Cancer by Mutations in β-Catenin or APCScience, 1997
- Nuclear localization of β-catenin by interaction with transcription factor LEF-1Mechanisms of Development, 1996
- Suppression of tumorigenicity by plakoglobin: an augmenting effect of N-cadherin.The Journal of cell biology, 1996
- Production of high-titer helper-free retroviruses by transient transfection.Proceedings of the National Academy of Sciences, 1993