The c-Jun NH2-terminal Kinase Promotes Insulin Resistance during Association with Insulin Receptor Substrate-1 and Phosphorylation of Ser307
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Open Access
- 1 March 2000
- journal article
- Published by Elsevier BV
- Vol. 275 (12), 9047-9054
- https://doi.org/10.1074/jbc.275.12.9047
Abstract
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This publication has 63 references indexed in Scilit:
- Interaction of Insulin Receptor Substrate-1 (IRS-1) with Phosphatidylinositol 3-Kinase: Effect of Substitution of Serine for Alanine in Potential IRS-1 Serine Phosphorylation SitesEndocrinology, 1998
- Protein Kinase C Modulates the Insulin-Stimulated Increase in Akt1 and Akt3 Activity in 3T3-L1 AdipocytesBiochemical and Biophysical Research Communications, 1998
- Tumor Necrosis Factor-αStroke, 1997
- Requirement of Phosphatidylinositol-3 Kinase for Activation of JNK/SAPKs by PDGFBiochemical and Biophysical Research Communications, 1997
- Inhibition of Tumor Necrosis Factor and Amelioration of Brain Infarction in MiceJournal of Cerebral Blood Flow & Metabolism, 1997
- jun N-terminal Kinase Mediates Activation of Skeletal Muscle Glycogen Synthase by Insulin in VivoPublished by Elsevier BV ,1996
- Activation of cJun NH2-Terminal Kinase/Stress-Activated Protein Kinase by InsulinBiochemistry, 1996
- Tumor Necrosis Factor α-induced Phosphorylation of Insulin Receptor Substrate-1 (IRS-1)Journal of Biological Chemistry, 1995
- Pro-inflammatory Cytokines and Environmental Stress Cause p38 Mitogen-activated Protein Kinase Activation by Dual Phosphorylation on Tyrosine and ThreonineJournal of Biological Chemistry, 1995
- Adipose Expression of Tumor Necrosis Factor-α: Direct Role in Obesity-Linked Insulin ResistanceScience, 1993