Downregulation of c-fos gene transcription in cells transformed by E1A and cHa-ras oncogenes: a role of sustained activation of MAP/ERK kinase cascade and of inactive chromatin structure at c-fos promoter
- 24 January 2002
- journal article
- Published by Springer Science and Business Media LLC in Oncogene
- Vol. 21 (5), 719-730
- https://doi.org/10.1038/sj.onc.1205118
Abstract
Oncogene is one of the world’s leading cancer journals. It is published weekly and covers all aspects of the structure and function of Oncogenes. Oncogene also publishes 8 Reviews issues a year, on a broad range of topics.Keywords
This publication has 55 references indexed in Scilit:
- F9 embryonal carcinoma cells fail to stop at G1/S boundary of the cell cycle after γ-irradiation due to p21WAF1/CIP1 degradationOncogene, 2000
- Activation of the c-fos enhancer by the Erk MAP kinase pathway through two sequence elements: the c-fos AP-1 and p62TCF sitesOncogene, 2000
- ERK activation induces phosphorylation of Elk-1 at multiple S/T-P motifs to high stoichiometryOncogene, 1999
- Deregulation of p53/p21Cip1/Waf1 pathway contributes to polyploidy and apoptosis of E1A+cHa-ras transformed cells after γ-irradiationOncogene, 1999
- One-way Cross-talk between p38MAPK and p42/44MAPKPublished by Elsevier BV ,1999
- Id helix–loop–helix proteins inhibit nucleoprotein complex formation by the TCF ETS-domain transcription factorsThe EMBO Journal, 1999
- Transformation by ras modifies AP1 composition and activityOncogene, 1997
- The Transcriptional Coactivators p300 and CBP Are Histone AcetyltransferasesCell, 1996
- In vivo protein‐DNA interactions at the c‐jun promoter in quiescent and serum‐stimulated fibroblastsJournal of Cellular Biochemistry, 1995
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976