PPARγ Mediates High-Fat Diet–Induced Adipocyte Hypertrophy and Insulin Resistance
Open Access
- 1 October 1999
- journal article
- research article
- Published by Elsevier BV in Molecular Cell
- Vol. 4 (4), 597-609
- https://doi.org/10.1016/s1097-2765(00)80210-5
Abstract
No abstract availableThis publication has 24 references indexed in Scilit:
- Orphan Nuclear Receptors: Shifting Endocrinology into ReverseScience, 1999
- ADD1/SREBP1 activates PPARγ through the production of endogenous ligandProceedings of the National Academy of Sciences of the United States of America, 1998
- Troglitazone increases the number of small adipocytes without the change of white adipose tissue mass in obese Zucker rats.JCI Insight, 1998
- Functional Antagonism between CCAAT/Enhancer Binding Protein-α and Peroxisome Proliferator-activated Receptor-γ on the Leptin PromoterPublished by Elsevier BV ,1997
- Thiazolidinediones in the Treatment of Insulin Resistance and Type II DiabetesDiabetes, 1996
- Antidiabetic thiazolidinediones inhibit leptin (ob) gene expression in 3T3-L1 adipocytes.Proceedings of the National Academy of Sciences of the United States of America, 1996
- High fat diet-induced hyperglycemia: prevention by low level expression of a glucose transporter (GLUT4) minigene in transgenic mice.Proceedings of the National Academy of Sciences of the United States of America, 1995
- Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factorCell, 1994
- Differential expression and activation of a family of murine peroxisome proliferator-activated receptors.Proceedings of the National Academy of Sciences of the United States of America, 1994
- A Novel ES Cell Line, TT2, with High Germline-Differentiating PotencyAnalytical Biochemistry, 1993