Protection of podocytes from hyperhomocysteinemia-induced injury by deletion of the gp91phox gene
- 15 April 2010
- journal article
- Published by Elsevier BV in Free Radical Biology & Medicine
- Vol. 48 (8), 1109-1117
- https://doi.org/10.1016/j.freeradbiomed.2010.01.029
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- RAGE Mediates Podocyte Injury in Adriamycin-induced GlomerulosclerosisJournal of the American Society of Nephrology, 2008
- Mechanisms of Homocysteine-Induced Glomerular Injury and SclerosisAmerican Journal of Nephrology, 2007
- Actin up: regulation of podocyte structure and function by components of the actin cytoskeletonTrends in Cell Biology, 2007
- Pitavastatin ameliorates albuminuria and renal mesangial expansion by downregulating NOX4 in db/db miceKidney International, 2007
- Acid Sphingomyelinase and Its Redox Amplification in Formation of Lipid Raft Redox Signaling Platforms in Endothelial CellsAntioxidants and Redox Signaling, 2007
- Podocyte Injury and Glomerulosclerosis in Hyperhomocysteinemic RatsAmerican Journal of Nephrology, 2007
- NADPH oxidase inhibition improves neurological outcomes in surgically-induced brain injuryNeuroscience Letters, 2007
- Gp91phox-containing NAD(P)H oxidase increases superoxide formation by doxorubicin and NADPHFree Radical Biology & Medicine, 2007
- Enhanced Aldosterone Signaling in the Early Nephropathy of Rats with Metabolic SyndromeJournal of the American Society of Nephrology, 2006
- Puromycin aminonucleoside induces oxidant-dependent DNA damage in podocytes in vitro and in vivoKidney International, 2006