Alzheimer Amyloid β-Peptide Inhibits the Late Phase of Long-Term Potentiation through Calcineurin-Dependent Mechanisms in the Hippocampal Dentate Gyrus
- 31 May 2002
- journal article
- Published by Elsevier BV in Neurobiology of Learning and Memory
- Vol. 77 (3), 354-371
- https://doi.org/10.1006/nlme.2001.4034
Abstract
No abstract availableKeywords
This publication has 64 references indexed in Scilit:
- Impairment of hippocampal long-term potentiation by Alzheimer amyloid ?-peptidesJournal of Neuroscience Research, 2000
- Impaired synaptic plasticity and learning in aged amyloid precursor protein transgenic miceNature Neuroscience, 1999
- Gating of CaMKII by cAMP-Regulated Protein Phosphatase Activity During LTPScience, 1998
- Regulatory Phosphorylation of AMPA-Type Glutamate Receptors by CaM-KII During Long-Term PotentiationScience, 1997
- Genetic Demonstration of a Role for PKA in the Late Phase of LTP and in Hippocampus-Based Long-Term MemoryCell, 1997
- CREB Phosphorylation and Dephosphorylation: A Ca2+- and Stimulus Duration–Dependent Switch for Hippocampal Gene ExpressionCell, 1996
- Postsynaptic CAMP pathway gates early LTP in hippocampal CA1 regionNeuron, 1995
- The Ca2+ influx induced by β‐amyloid peptide 25–35 in cultured hippocampal neurons results from network excitationJournal of Neurobiology, 1995
- Mechanisms underlying induction of long-term potentiation in rat medial and lateral perforant paths in vitroJournal of Neurophysiology, 1993
- An age comparison of the rates of acquisition and forgetting of spatial information in relation to long-term enhancement of hippocampal synapses.Behavioral Neuroscience, 1985