ERK1/2 inactivation and p38 MAPK-dependent caspase activation during guanosine 5′-triphosphate-mediated terminal erythroid differentiation of K562 cells
- 31 December 2007
- journal article
- Published by Elsevier BV in The International Journal of Biochemistry & Cell Biology
- Vol. 39 (9), 1685-1697
- https://doi.org/10.1016/j.biocel.2007.04.016
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Cyclic nucleotide response element binding (CREB) protein activation is involved in K562 erythroleukemia cells differentiationJournal of Cellular Biochemistry, 2006
- Activin A induces erythroid gene expressions and inhibits mitogenic cytokine‐mediated K562 colony formation by activating p38 MAPKJournal of Cellular Biochemistry, 2006
- Differentiation therapy of cancer. Potential advantages over conventional therapeutic approaches targeting death of cancer/tumor cellsMedical Hypotheses, 2005
- Effects of anthracycline derivatives on human leukemia K562 cell growth and differentiationBiochemical Pharmacology, 2005
- Bcr-Abl-Mediated Protection from Apoptosis Downstream of Mitochondrial Cytochrome c ReleaseMolecular and Cellular Biology, 2004
- Chronic myelogenous leukemia as a paradigm of early cancer and possible curative strategiesLeukemia, 2003
- Inducible p27Kip1 expression inhibits proliferation of K562 cells and protects against apoptosis induction by proteasome inhibitorsCell Death & Differentiation, 2003
- p53 expression in K562 cells is associated with caspase‐mediated cleavage of c‐ABL and BCR–ABL protein kinasesBritish Journal of Haematology, 2002
- Apoptosis-associated release of Smac/DIABLO from mitochondria requires active caspases and is blocked by Bcl-2The EMBO Journal, 2001
- Function of caspases in regulating apoptosis caused by erythropoietin deprivation in erythroid progenitorsJournal of Cellular Physiology, 1999