Apoptosis Repressor with Caspase Recruitment Domain Protects against Cell Death by Interfering with Bax Activation
Open Access
- 1 May 2004
- journal article
- Published by Elsevier BV
- Vol. 279 (20), 21233-21238
- https://doi.org/10.1074/jbc.m400695200
Abstract
No abstract availableThis publication has 37 references indexed in Scilit:
- Inducible Expression of BNIP3 Provokes Mitochondrial Defects and Hypoxia-Mediated Cell Death of Ventricular MyocytesCirculation Research, 2002
- TAT Protein Transduction Into Isolated Perfused HeartsCirculation, 2002
- Signaling Pathways in Reactive Oxygen Species–Induced Cardiomyocyte ApoptosisCirculation, 1999
- Enhanced expression of p53 and apoptosis induced by blockade of the vacuolar proton ATPase in cardiomyocytes.JCI Insight, 1998
- The liver isoform of carnitine palmitoyltransferase I is activated in neonatal rat cardiac myocytes by hypoxiaMolecular and Cellular Biochemistry, 1998
- Apoptosis in the Failing Human HeartThe New England Journal of Medicine, 1997
- Apoptosis in Human Acute Myocardial InfarctionCirculation, 1997
- Apoptosis in Myocytes in End-Stage Heart FailureThe New England Journal of Medicine, 1996
- Expression of bcl-2 Protein, an Inhibitor of Apoptosis, and Bax, an Accelerator of Apoptosis, in Ventricular Myocytes of Human Hearts With Myocardial InfarctionCirculation, 1996
- Reperfusion injury induces apoptosis in rabbit cardiomyocytes.JCI Insight, 1994