Cyclooxygenase-2-derived Prostaglandin E2 Promotes Human Cholangiocarcinoma Cell Growth and Invasion through EP1 Receptor-mediated Activation of the Epidermal Growth Factor Receptor and Akt
Open Access
- 1 June 2005
- journal article
- research article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 280 (25), 24053-24063
- https://doi.org/10.1074/jbc.m500562200
Abstract
No abstract availableThis publication has 44 references indexed in Scilit:
- Nail-Biting Time for Trials of COX-2 DrugsScience, 2004
- Sustained activation of epidermal growth factor receptor in cholangiocarcinoma: a potent therapeutic target?Journal of Hepatology, 2004
- Vioxx Withdrawal Alarms Cancer Prevention ResearchersJNCI Journal of the National Cancer Institute, 2004
- Enhanced epidermal growth factor receptor activation in human cholangiocarcinoma cellsJournal of Hepatology, 2004
- The Molecular Pathogenesis of CholangiocarcinomaSeminars in Liver Disease, 2004
- Cholangiocarcinoma: Molecular targeting strategies for chemoprevention and therapyHepatology, 2004
- PPARγ ligands inhibit cholangiocarcinoma cell growth through p53-dependent GADD45 and p21WAF1/Cip1 pathwayHepatology, 2003
- Differential expression of cyclooxygenase-2 (COX-2) in human bile duct epithelial cells and bile duct neoplasmHepatology, 2001
- Epidermal growth factor receptor signalingCurrent Biology, 2001
- Aberrant cyclooxygenase isozyme expression in human intrahepatic cholangiocarcinomaGut, 2001