Elimination of Myostatin Does Not Combat Muscular Dystrophy in dy Mice but Increases Postnatal Lethality
- 28 February 2005
- journal article
- Published by Elsevier BV in The American Journal of Pathology
- Vol. 166 (2), 491-497
- https://doi.org/10.1016/s0002-9440(10)62271-7
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Highly effective SNP-based association mapping and management of recessive defects in livestockNature Genetics, 2008
- Skeletal Muscle Signaling Pathway through the Dystrophin Glycoprotein Complex and Rac1Published by Elsevier BV ,2003
- Pharmacological strategies for muscular dystrophyNature Reviews Drug Discovery, 2003
- Spontaneous muscular dystrophy caused by a retrotransposal insertion in the mouse laminin α2 chain geneNeuromuscular Disorders, 2003
- Merosin-deficient congenital muscular dystrophy, autosomal recessive (MDC1A, MIM#156225, LAMA2 gene coding for α2 chain of laminin)European Journal of Human Genetics, 2002
- An agrin minigene rescues dystrophic symptoms in a mouse model for congenital muscular dystrophyNature, 2001
- Disruption of thelama2Gene in Embryonic Stem Cells: Laminin α2 Is Necessary for Sustenance of Mature Muscle CellsExperimental Cell Research, 1998
- A deletion in the bovine myostatin gene causes the double–muscled phenotype in cattleNature Genetics, 1997
- Regulation of skeletal muscle mass in mice by a new TGF-p superfamily memberNature, 1997
- Muscular dystrophy in the mouse caused by an allele at the dy-locusLife Sciences, 1970