Expression of γ-glutamyltransferase in cancer cells and its significance in drug resistance
- 12 January 2006
- journal article
- review article
- Published by Elsevier BV in Biochemical Pharmacology
- Vol. 71 (3), 231-238
- https://doi.org/10.1016/j.bcp.2005.10.005
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- Endogenous Oxidative Stress Induces Distinct Redox Forms of Tumor Necrosis Factor Receptor-1 in Melanoma CellsAnnals of the New York Academy of Sciences, 2004
- γ-Glutamyl transpeptidase catalyses the extracellular detoxification of cisplatin in a human cell line derived from the proximal convoluted tubule of the kidneyEuropean Journal of Cancer, 2003
- γ-glutamyltransferase dependent generation of reactive oxygen species from a glutathione/transferrin systemFree Radical Biology & Medicine, 1998
- Increased lipoperoxide levels and antioxidant system in colorectal cancerZeitschrift für Krebsforschung und Klinische Onkologie, 1998
- The cell-specific anti-proliferative effect of reduced glutathione is mediated by γ-glutamyl transpeptidase-dependent extracellular pro-oxidant reactionsInternational Journal of Cancer, 1997
- -Glutamyl Transpeptidase Mediation of Tumor Glutathione Utilization In VivoJNCI Journal of the National Cancer Institute, 1996
- γ-Glutamyl transpeptidase-cellular expression in populations of normal human mononuclear cells and patients suffering from leukemiasAnnals of Hematology, 1995
- Glutathione stimulates A549 cell proliferation in glutamine‐deficient culture: The effect of glutamate supplementationJournal of Cellular Physiology, 1994
- Glutathione metabolism by γ-glutamyltranspeptidase leads to lipid peroxidation: characterization of the system and relevance to hepatocarcinogenesisCarcinogenesis: Integrative Cancer Research, 1993
- Protective effect of reduced glutathione against cisplatin-induced renal and systemic toxicity and its influence on the therapeutic activity of the antitumor drugChemico-Biological Interactions, 1989