Parathyroid hormone receptor signaling in osteocytes increases the expression of fibroblast growth factor-23 in vitro and in vivo
- 31 October 2011
- journal article
- Published by Elsevier BV in Bone
- Vol. 49 (4), 636-643
- https://doi.org/10.1016/j.bone.2011.06.025
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (R01 DK076007, R03 TW06919, R01 AR053643, DK063934)
- Lilly Endowment, Inc
- Yonsei University, South Korea
This publication has 42 references indexed in Scilit:
- Fibroblast growth factor 23 is elevated before parathyroid hormone and phosphate in chronic kidney diseaseKidney International, 2011
- PTH receptor signaling in osteocytes governs periosteal bone formation and intracortical remodelingJournal of Bone and Mineral Research, 2010
- Control of RANKL gene expressionBone, 2010
- Bone Formation Regulates Circulating Concentrations of Fibroblast Growth Factor 23Endocrinology, 2009
- Novel Regulators of Fgf23 Expression and Mineralization in Hyp BoneMolecular Endocrinology, 2009
- Genetic Evidence of Serum Phosphate-Independent Functions of FGF-23 on BonePLoS Genetics, 2008
- Amelioration of the premature ageing‐like features of Fgf‐23 knockout mice by genetically restoring the systemic actions of FGF‐23The Journal of Pathology, 2008
- The parathyroid is a target organ for FGF23 in ratsJCI Insight, 2007
- Klotho converts canonical FGF receptor into a specific receptor for FGF23Nature, 2006
- Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolismNature Genetics, 2006