Distinct Signaling Requirements for Dμ Selection, IgH Allelic Exclusion, Pre-B Cell Transition, and Tumor Suppression in B Cell Progenitors
- 1 June 2003
- journal article
- Published by Elsevier BV in Immunity
- Vol. 18 (6), 825-836
- https://doi.org/10.1016/s1074-7613(03)00142-0
Abstract
No abstract availableThis publication has 65 references indexed in Scilit:
- Control of Pre-BCR Signaling by Pax5-Dependent Activation of the BLNK GeneImmunity, 2002
- Truncated immunoglobulin Dμ causes incomplete developmental progression of RAG-deficient pro-B cellsMolecular Immunology, 2002
- Interference with Immunoglobulin (Ig)α Immunoreceptor Tyrosine–Based Activation Motif (Itam) Phosphorylation Modulates or Blocks B Cell Development, Depending on the Availability of an Igβ Cytoplasmic TailThe Journal of Experimental Medicine, 2001
- A Prematurely Expressed Igκ Transgene, but Not a VκJκ Gene Segment Targeted into the Igκ Locus, Can Rescue B Cell Development in λ5-Deficient MiceImmunity, 1996
- Assembly of the Truncated Immunoglobulin Heavy Chain Dμ into Antigen Receptor–Like Complexes in Pre-B Cells but Not in B CellsImmunity, 1996
- Defective B cell development and function in Btk-deficient miceImmunity, 1995
- Analysis of the B-cell progenitor compartment at the level of single cellsCurrent Biology, 1994
- Virus-transformed pre-B cells show ordered activation but not inactivation of immunoglobulin gene rearrangement and transcription.The Journal of Experimental Medicine, 1991
- Feedback inhibition of immunoglobulin gene rearrangement by membrane mu, but not by secreted mu heavy chains.The Journal of Experimental Medicine, 1988
- Allelic Exclusion in Transgenic Mice That Express the Membrane form of Immunoglobulin μScience, 1987