Voluntary exercise protects hippocampal neurons from trimethyltin injury: Possible role of interleukin-6 to modulate tumor necrosis factor receptor-mediated neurotoxicity
- 22 March 2011
- journal article
- Published by Elsevier BV in Brain, Behavior, and Immunity
- Vol. 25 (6), 1063-1077
- https://doi.org/10.1016/j.bbi.2011.03.012
Abstract
No abstract availableKeywords
This publication has 71 references indexed in Scilit:
- In humans IL‐6 is released from the brain during and after exercise and paralleled by enhanced IL‐6 mRNA expression in the hippocampus of miceActa Physiologica, 2010
- Hypothalamic Pituitary Adrenal Axis Responses to Low‐Intensity Stressors are Reduced After Voluntary Wheel Running in RatsJournal of Neuroendocrinology, 2010
- Short-term exercise in aged Tg2576 mice alters neuroinflammation and improves cognitionNeurobiology of Disease, 2008
- Exercise alters the immune profile in Tg2576 Alzheimer mice toward a response coincident with improved cognitive performance and decreased amyloidJournal of Neuroinflammation, 2008
- STAT dynamicsCytokine & Growth Factor Reviews, 2007
- Unphosphorylated STAT3 accumulates in response to IL-6 and activates transcription by binding to NFκBGenes & Development, 2007
- Activation of N-ras and K-ras induced by interleukin-6 in a myeloma cell line: implications for disease progression and therapeutic responseOncogene, 2002
- Induction of STAT3 Signaling in Activated Astrocytes and Sprouting Septal Neurons Following Entorhinal Cortex Lesion in Adult RatsMolecular and Cellular Neuroscience, 2002
- Tissue Expression and Plasma Concentrations of TNFα, IL-1β, and IL-6 Following Treadmill Exercise in MiceInternational Journal of Sports Medicine, 2001
- Ramification of microglia, monocytes and macrophages in vitro: influences of various epithelial and mesenchymal cells and their conditioned media *Cell and tissue research, 1997