Recruitment of activated IRF-3 and CBP/p300 to herpes simplex virus ICP0 nuclear foci: Potential role in blocking IFN-β induction
- 28 November 2006
- journal article
- Published by Elsevier BV in Virology
- Vol. 360 (2), 305-321
- https://doi.org/10.1016/j.virol.2006.10.028
Abstract
No abstract availableKeywords
This publication has 96 references indexed in Scilit:
- The protein ICP0 of herpes simplex virus type 1 is targeted to nucleoli of infected cellsArchiv für die gesamte Virusforschung, 2005
- Mechanism for transcriptional synergy between interferon regulatory factor (IRF)-3 and IRF-7 in activation of the interferon-β gene promoterEuropean Journal of Biochemistry, 2004
- Toll-like Receptor 9–mediated Recognition of Herpes Simplex Virus-2 by Plasmacytoid Dendritic CellsThe Journal of Experimental Medicine, 2003
- Interferon regulatory factor-3 is an in vivo target of DNA-PKProceedings of the National Academy of Sciences of the United States of America, 2002
- Downregulation of IRF-3 Levels by Ribozyme Modulates the Profile of IFNA Subtypes Expressed in Infected Human CellsJournal of Virology, 2001
- Herpes Simplex Virus Triggers and Then Disarms a Host Antiviral ResponseJournal of Virology, 2001
- Involvement of the IRF family transcription factor IRF‐3 in virus‐induced activation of the IFN‐β geneFEBS Letters, 1998
- Functional Role of Type I and Type II Interferons in Antiviral DefenseScience, 1994
- Alternative Mechanisms for Activation of Human Immunodeficiency Virus Enhancer in T CellsScience, 1988
- Identification of herpes simplex virus DNA sequences which encode a trans-acting polypeptide responsible for stimulation of immediate early transcriptionJournal of Molecular Biology, 1984