Kainic acid induces selective mitochondrial oxidative phosphorylation enzyme dysfunction in cerebellar granule neurons: protective effects of melatonin and GSH ethyl ester
Open Access
- 18 June 2001
- journal article
- Published by Wiley in The FASEB Journal
- Vol. 15 (10), 1786-1788
- https://doi.org/10.1096/fj.00-0427fje
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- The role of intracellular Na+ and mitochondria in buffering of kainate‐induced intracellular free Ca2+ changes in rat forebrain neuronesThe Journal of Physiology, 1998
- Striatal Malonate Lesions Are Attenuated in Neuronal Nitric Oxide Synthase Knockout MiceJournal of Neurochemistry, 1996
- In vitro and in vivo protection against kainate‐induced excitotoxicity by melatoninJournal of Pineal Research, 1996
- Role of mitochondria in the etiology and pathogenesis of Parkinson's diseaseBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1995
- Melatonin protects primary cultures of cerebellar granule neurons from kainate but not from N-methyl-d-aspartate excitotoxicityExperimental Neurology, 1995
- Oxidative Stress, Glutamate, and Neurodegenerative DisordersScience, 1993
- Degeneration of hippocampal CA3 pyramidal cells induced by intraventricular kainic acidJournal of Comparative Neurology, 1980
- Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications.Proceedings of the National Academy of Sciences of the United States of America, 1979
- In situ injection of kainic acid: A new method for selectively lesioning neuronal cell bodies while sparing axons of passageJournal of Comparative Neurology, 1978
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976