The serum 24,25-dihydroxyvitamin D concentration, a marker of vitamin D catabolism, is reduced in chronic kidney disease
Open Access
- 1 September 2012
- journal article
- Published by Elsevier BV in Kidney International
- Vol. 82 (6), 693-700
- https://doi.org/10.1038/ki.2012.193
Abstract
No abstract availableThis publication has 43 references indexed in Scilit:
- Quantification of 1α,25-Dihydroxy Vitamin D by Immunoextraction and Liquid Chromatography–Tandem Mass SpectrometryClinical Chemistry, 2011
- Changes in bone mineral parameters, vitamin D metabolites, and PTH measurements with varying chronic kidney disease stagesJournal of Bone and Mineral Metabolism, 2010
- A rubber transfer gasket to improve the throughput of liquid–liquid extraction in 96-well plates: Application to vitamin D testingJournal of Chromatography B, 2010
- Standards of Medical Care in Diabetes—2010Diabetes Care, 2010
- Mitochondrial dysregulation and oxidative stress in patients with chronic kidney diseaseBMC Genomics, 2009
- Reduction of the vitamin D hormonal system in kidney disease is associated with increased renal inflammationKidney International, 2008
- Mineral metabolism parameters throughout chronic kidney disease stages 1-5--achievement of K/DOQI target rangesNephrology Dialysis Transplantation, 2007
- FGF-23 Is a Potent Regulator of Vitamin D Metabolism and Phosphate HomeostasisJournal of Bone and Mineral Research, 2004
- Human Pharmacokinetics of Orally Administered (24 R)-Hydroxycalcidiolcclm, 1993
- Marked suppression of secondary hyperparathyroidism by intravenous administration of 1,25-dihydroxy-cholecalciferol in uremic patients.JCI Insight, 1984